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Miners, J., Chalmers, K., Kehoe, P., Love, S., Helmond, Z. and Wilcock, G. (2006) Decreased expression and activity of neprilysin in Alzheimer disease are associated with cerebral amyloid angiopathy. Journal of Neuropathology & Experimental Neurology, 65 (10). pp. 1012-1021. ISSN 0022-3069 Available from: http://eprints.uwe.ac.uk/1518
Chalmers, K., Love, S. and Wilcock, G. (2005) Contributors to white matter damage in the frontal lobe in Alzheimer's disease. Neuropathology and applied neurobiology, 31 (6). pp. 623-631. ISSN 0305-1846 Available from: http://eprints.uwe.ac.uk/1514
Chalmers, K., Love, S., Williams, S. and Wilcock, G. (2005) Relationship of neurofibrillary pathology to cerebral amyloid angiopathy in Alzheimer's disease. Neuropathology and applied neurobiology, 31 (4). pp. 414-421. ISSN 0305-1846 Available from: http://eprints.uwe.ac.uk/1619
Chalmers, K., Blomqvist, M.-L., Andreasen, N., Bogdanovic, N. and Wilcock, G. (2005) Sequence variants of IDE are associated with the extent of beta-amyloid deposition in the Alzheimer's disease brain. Neurobilogy of Aging, 26 (6). pp. 795-802. ISSN 0197-4580 Available from: http://eprints.uwe.ac.uk/1512
Chalmers, K., Wilcock, G. and Love, S. (2003) APOEɛ4 influences the pathological phenotype of Alzheimer's disease by favouring cerebrovascular over parenchymal accumulation of Aβ protein. Neuropathology and Applied Neurobiology, 29 (3). pp. 231-238. ISSN 0305-1846 Available from: http://eprints.uwe.ac.uk/5074
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